Yazar "Gökçe, Burcu" seçeneğine göre listele

Listeleniyor 1 - 4 / 4
  • Küçük Resim
    Öğe
    Pregabalin-Induced Akathisia
    (Sage Publications Inc, 2013) Dağ, Ersel; Gökçe, Burcu; Buturak, Sadiye Visal; Tiryaki, Duygu; Erdemoğlu, Ali Kemal
    …
  • Küçük Resim
    Öğe
    Rare side effects of valproic acid: Gingival hyperplasia, and cervical lymphadenopathy
    (2013) Dağ, Ersel; Demirer, Serhat ; Gökçe, Burcu ; Acar, Betül; Keçel, Hüseyin Gencal; Aylıkçı, Uğur Bahadır; Erdemoğlu, Ali Kemal
    Gingival hyperplasia is a common disorder seen in patients with epilepsy treated with phenytoin. It is rarely seen in patients taking sodium valproate therapy. Especially in mentally retarded patients, poor oral hygiene causes to the accumulation of bacterial plaque and inflammation might be occurs. Connective tissue increases due to inflammation and gingival hyperplasia is seen. We report a case of gingival hyperplasia due to valproic acid use to draw attention to rare adverse effect of this drug.
  • [ X ]
    Öğe
    Sneddon’s syndrome presenting with neuropathic pain
    (Pulse Marketing and Communications LLC, 2013) Dağ, Ersel; Gökçe, Burcu; Koçak, Mukadder
    A 55-year-old man was admitted to us with a sense of numbness, tingling, and burning in his feet and headache, characterized as a feeling of pressure all around his head, for 1 year and aggravated in the past 3 months. The patient’s neurologic examination was normal and he had no other known diseases except for hypertension according to his medical history. During the examination, we recognized purplish lesions on the patient’s body. His kidney, liver, and thyroid function test results and vitamin B12 levels were all normal. His hematocrit level was 41.8%, platelet value was 234,000 (150,000-500,000), and sedimentation rate was 9 mm/h (0-20). Electromyography was performed and results were found to be normal. The patient was diagnosed as having small fiber neuropathy. Dermatologic examination revealed reddish blue mottling of the skin with fishnet reticular pattern on his back, on the front side of the body, and on both arms and legs, and the lesions were classified as livedo racemosa (Figure 1). Brain magnetic resonance imaging (MRI) showed subcortical hyperintense ischemic-gliotic signal changes on T2-FLAIR in the deep white matter of bilateral frontoparietal vertex, centrum semiovale, and corona radiata (Figure 2). FLAIR sequence axial MRI of the brain of our patient showed subcortical hyperintense lesions in both cerebral hemispheres. His cardiac examination was normal and minimal aortic regurgitation was seen on echocardiography. His cognitive assessment Minimental Test Score was 22, and Montreal Cognitive Assessment score was 18. Laboratory values for inflammatory markers and autoimmune antibodies including syphilis serology, lupus anticoagulants, and anticardiolipin antibodies were negative. Factor V Leiden mutation was not detected in the patient. The patient was diagnosed with Sneddon’s syndrome with the above signs and symptoms and small fiber neuropathy. Clopidogrel 75 mg and gabapentin 1200 mg was started once a day and blood pressure regulation was achieved. © 2013 Pulse Marketing & Communications, LLC.
  • Küçük Resim
    Öğe
    Status Epilepticus in a Patient with Amyotrophic Lateral Sclerosis
    (Aves, 2014) Dağ, Ersel; Şahin, Oruç; Gökçe, Burcu; Erdemoğlu, Ali Kemal
    Introduction: Amyotrophic Lateral Sclerosis, the most common and well-known form of motor neuron disease, is characterized by progressive degeneration of upper and lower motor neurons. Respiratory failure and aspiration due to respiratory muscle weakness is the most common causes of mortality in patients with amyotrophic lateral sclerosis. Status epilepticus is the second most frequent neurological emergency with a risk of major morbidity or mortality. Status epilepticus, due to causes such as prolonged seizures, respiratory distress, acidosis, hypoglycemia, and hypotension may lead to mortality. Case Report: A forty-eight year old male patient had been followed for 4 years with a diagnosis of amyotrophic lateral sclerosis and was referred to our clinic with the diagnosis of status epilepticus. The refractory status epilepticus patient was treated with a diagnosis of respiratory failure and the need for mechanical ventilation evolved. Conclusion: We report the development of status epilepticus in a patient with amyotrophic lateral sclerosis, and in addition to the appropriate anti-epileptic medication, ventilator support and intensive care was needed, emphasizing the life-saving procedure.