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    Carotid Artery Dissection and Cerebral Infarction Secondary to Blunt Trauma
    (Turkish Neurological Soc, 2012) Gokce, Burcu; Erdemoglu, A. Kemal
    Traumatic carotid artery dissection may appear after blunt head or neck traumas. Patients can be asymptomatic or clinical symptoms may include headache, transient ischemic attack, stroke, loss of consciousness, hemiparesis, aphasia and Horner syndrome, typically occurring within hours to days. Prognosis is good if diagnosed and treated early. As cerebral ischemia and neurological deficits may develop subsequently, it is essential that carotid artery dissection should be considered, and possible cases evaluated and diagnosed appropriately. In this article, we present a case of internal carotid artery dissection with diagnostic neuro-imaging findings, that occurred after 6 hours of blunt trauma and subsequent cerebral infarction following a car accident. Clinical features, neuro-radiological diagnostic methods and treatments options are discussed with relevant literature.
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    Colchicine-Related Polyneuropathy and Multiple Organ Failure
    (Turkish Neurological Soc, 2013) Dag, Ersel; Turkel, Yakup; Gokce, Burcu
    Colchicine arrests microtubule assembly and inhibits many cellular functions. This drug is used to treat gout and and Familial Mediterranean Fever. Its gastrointestinal side effects are frequent but more severe adverse effects such as neuropathy and multi-organ failure associated with colchicine are rare. The blood levels of colchicine are dependent on the liver and kidneys. Thus renal or liver failure can result in colchicine toxication. We presented a case of colchicine toxicity, which resulted in multi-organ failure and polyneuropathy.
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    Status Epilepticus in a Patient with Amyotrophic Lateral Sclerosis
    (Aves, 2014) Dag, Ersel; Sahin, Oruc; Gokce, Burcu; Erdemoglu, Ali Kemal
    Introduction: Amyotrophic Lateral Sclerosis, the most common and well-known form of motor neuron disease, is characterized by progressive degeneration of upper and lower motor neurons. Respiratory failure and aspiration due to respiratory muscle weakness is the most common causes of mortality in patients with amyotrophic lateral sclerosis. Status epilepticus is the second most frequent neurological emergency with a risk of major morbidity or mortality. Status epilepticus, due to causes such as prolonged seizures, respiratory distress, acidosis, hypoglycemia, and hypotension may lead to mortality. Case Report: A forty-eight year old male patient had been followed for 4 years with a diagnosis of amyotrophic lateral sclerosis and was referred to our clinic with the diagnosis of status epilepticus. The refractory status epilepticus patient was treated with a diagnosis of respiratory failure and the need for mechanical ventilation evolved. Conclusion: We report the development of status epilepticus in a patient with amyotrophic lateral sclerosis, and in addition to the appropriate anti-epileptic medication, ventilator support and intensive care was needed, emphasizing the life-saving procedure.