Periodontal health in children exposed to passive smoking

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dc.contributor.authorErdemir, Ebru Olgun
dc.contributor.authorSönmez, Isil Saroglu
dc.contributor.authorOba, Aylin Akbay
dc.contributor.authorBergstrom, Jan
dc.contributor.authorÇağlayan, Osman
dc.date.accessioned2020-06-25T17:51:18Z
dc.date.available2020-06-25T17:51:18Z
dc.date.issued2010
dc.departmentKırıkkale Üniversitesi
dc.descriptionsonmez, isil/0000-0002-3530-0244
dc.description.abstractP>Aim To determine (1) the cotinine levels of saliva, urine and gingival crevicular fluid (GCF) of children in families with and without smoking members and (2) a possible association between the periodontal health of the children and exposure to passive smoking. Material and Methods The study population comprised of 109 children in the age range 6-12 years. Children were classified as exposed to passive tobacco smoking (PTS-exposed, n=51) and as unexposed controls (PTS-unexposed, n=58). Plaque index, gingival index, bleeding on probing, probing depth and clinical attachment level (CAL) were recorded. GCF, saliva and urine samples were also collected. The levels of cotinine in these fluids were determined by enzyme-linked immunosorbent assay. Results The mean salivary cotinine concentration was significantly increased in PTS-exposed children compared with PTS-unexposed children (p < 0.05). Further, in a dose-dependent way, the mean salivary concentration was significantly higher in children whose father or mother was a smoker (p < 0.05) as compared, respectively, with children whose fathers and mothers were non-smokers. The mean CAL was significantly less in PTS-exposed children compared with non-PTS-exposed children (0.09 mm; p < 0.05) and also in children whose father was a smoker (p < 0.05), but not in children whose mother was a smoker as compared with non-smoker fathers and mothers, respectively. The GCF cotinine levels were below the detection limits with the assay method that was used. Conclusions We have observed that children who are exposed to passive smoking have elevated cotinine levels in their saliva concomitant with a lowered CAL.en_US
dc.identifier.citationclosedAccessen_US
dc.identifier.doi10.1111/j.1600-051X.2009.01510.x
dc.identifier.endpage164en_US
dc.identifier.issn0303-6979
dc.identifier.issn1600-051X
dc.identifier.issue2en_US
dc.identifier.pmid20041979
dc.identifier.scopus2-s2.0-74049120380
dc.identifier.scopusqualityQ1
dc.identifier.startpage160en_US
dc.identifier.urihttps://doi.org/10.1111/j.1600-051X.2009.01510.x
dc.identifier.urihttps://hdl.handle.net/20.500.12587/4798
dc.identifier.volume37en_US
dc.identifier.wosWOS:000273451300007
dc.identifier.wosqualityQ1
dc.indekslendigikaynakWeb of Science
dc.indekslendigikaynakScopus
dc.indekslendigikaynakPubMed
dc.language.isoen
dc.publisherWileyen_US
dc.relation.ispartofJournal Of Clinical Periodontology
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subjectchildrenen_US
dc.subjectcotinineen_US
dc.subjectpassive smokingen_US
dc.subjectperiodontal healthen_US
dc.titlePeriodontal health in children exposed to passive smokingen_US
dc.typeArticle

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